Subversion of Immune Cell Signalling by Parasites: Volume 41, Symposia of the British Society for Parasitology
One of the most studied fields within the discipline of parasitology is immunoparasitology. The main emphasis is understanding the interaction between the host immune system and the invading parasite in order that information can be gained to aid parasite control and hence improve human health. The articles contained within this volume were produced by speakers (and their colleagues) at the 2004 British Society for Parasitology Autumn Symposium on 'Subversion of immune cell signalling by parasites'. Together they provide an introduction to the topic as well as detailed insights into the immune subversion mechanisms employed by eukaryotic parasites.
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Subversion of immune cell signal Modulation of intracellular signal
List of contributions v Concluding remarks
Host antiviral innate immune response SI 3
Final remarks S20 contributions
acid adaptor AKIRA ALCAMI amastigotes antigen apoptosis binding Biology c-Myc Ca2+ cascade caspase cell lines cell signalling cellular CK2 activity CK2A complex cruzi cytokine degradation deletion Dessauge dsRNA encoded enzyme European Journal Experimental Medicine expression falciparum function gene induction glycan glycosylphosphatidylinositol GPI-anchored Harnett Heussler host cell IFN-y IFNy immune response immune system inactivation induced infected cells inhibition innate immune interaction interferon interleukin-1 intracellular Journal of Immunology Journal of Virology Leishmania donovani lipid lymphocytes macrophages malaria MAP kinase MAPK mechanisms mediated membrane MHC class mice modulation Molecular molecules mRNA murine NF-KB NFKB NFKB activation Olivier parasite Parasitology pathogens phosphatase phosphorylation Plasmodium poxvirus poxviruses PP2A production promastigotes protein kinase PTEN recruitment regulation Reiner replication role Schofield signal transduction signalling pathways SMITH subunit synthesis target Theileria TLR4 TLRs TNF-a Toll-like receptor TRAF6 transcription factors triggered tyrosine vaccinia virus viral viruses vitro VV infection