PharmacologyThe aim of this work is not only to describe what drugs do, but to emphasize the mechanisms by which they act - where possible at the cellular and molecular level. Therapeutic agents have a high rate of obsolescence and many new ones are introduced each year; an appreciation of the mechanisms of action of the class of drugs to which a new agent belongs provides a starting point for understanding and using intelligently. |
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Pagina 387
This explains why oral glucose causes greater insulin release than does
intravenous glucose . Such GIT hormones ( in particular GIP and GLP ) provide
an anticipatory signal from the GIT to the islets . Insulin release is inhibited by
several ...
This explains why oral glucose causes greater insulin release than does
intravenous glucose . Such GIT hormones ( in particular GIP and GLP ) provide
an anticipatory signal from the GIT to the islets . Insulin release is inhibited by
several ...
Pagina 389
stimulates cell proliferation and is implicated in somatic and visceral growth and
development . domain with phosphorylated IRS has several important effects
including recruitment of insulin - sensitive glucose transporters ( GLUT - 4 ) from
the ...
stimulates cell proliferation and is implicated in somatic and visceral growth and
development . domain with phosphorylated IRS has several important effects
including recruitment of insulin - sensitive glucose transporters ( GLUT - 4 ) from
the ...
Pagina 394
It acts more rapidly but for a shorter time than natural insulin , a feature that
enables patients to inject themselves soon before the start of a meal . this
possibility to avoid the mistake of increasing ( rather than reducing ) the dose of
insulin in this ...
It acts more rapidly but for a shorter time than natural insulin , a feature that
enables patients to inject themselves soon before the start of a meal . this
possibility to avoid the mistake of increasing ( rather than reducing ) the dose of
insulin in this ...
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Sommario
molecular aspects | 19 |
Method and measurement in pharmacology | 47 |
Absorption and distribution of drugs | 61 |
Copyright | |
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acetylcholine acid action activity agents agonists animals antagonists associated binding block blood body bone brain calcium cardiac cause cells central changes channels Chapter chemical clinical compounds concentration decrease dependence depression discussed disease dopamine dose drugs effects enzyme evidence example excretion factor function GABA gene given glucose glutamate growth half-life heart hormone human important increase inhibition inhibitors injection insulin interaction involved known less liver mainly mechanisms mediators membrane molecules myocardial infarction Nature nerve nervous system neurons noradrenaline normal occur oestrogens orally pain pathway patients peptides pharmacological phase plasma potential pressure prevent produce protein reactions receptors reduced regulation release response result risk role secretion selective shown similar smooth muscle specific stimulation structure studies substances synthesis Table terminals therapeutic therapy thyroid tissues transmission transmitter transport treat treatment trials types unwanted effects various vascular
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Informazioni bibliografiche
| Titolo | Pharmacology Churchill Livingstone |
| Autori | H. P. Rang, M. Maureen Dale, J. M. Ritter |
| Edizione | 4, illustrata, ristampa |
| Editore | Churchill Livingstone, 1999 |
| Provenienza dell'originale | la University of Michigan |
| Digitalizzato | 18 ago 2008 |
| ISBN | 0443059748, 9780443059742 |
| Lunghezza | 830 pagine |
| Esporta citazione | BiBTeX EndNote RefMan |