PharmacologyThe aim of this work is not only to describe what drugs do, but to emphasize the mechanisms by which they act - where possible at the cellular and molecular level. Therapeutic agents have a high rate of obsolescence and many new ones are introduced each year; an appreciation of the mechanisms of action of the class of drugs to which a new agent belongs provides a starting point for understanding and using intelligently. |
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Pagina 259
The pathology is basically the same as that involved in myocardial infarction ,
namely platelet - fibrin thrombus associated with a ruptured atheromatous plaque
, but without complete occlusion of the vessel . The risk of infarction is substantial
...
The pathology is basically the same as that involved in myocardial infarction ,
namely platelet - fibrin thrombus associated with a ruptured atheromatous plaque
, but without complete occlusion of the vessel . The risk of infarction is substantial
...
Pagina 260
... cardiac myocytes , have been disappointing , although they have chronic
treatment with captopril and other angiotensinconverting enzyme inhibitors
improves survival and haemodynamics in rat models of myocardial infarction ,
and several ...
... cardiac myocytes , have been disappointing , although they have chronic
treatment with captopril and other angiotensinconverting enzyme inhibitors
improves survival and haemodynamics in rat models of myocardial infarction ,
and several ...
Pagina 324
Uses mainly relate to arterial thrombosis , and include : • acute myocardial
infarction high risk of myocardial infarction , including : – patients who have
recovered from myocardial infarction - patients with symptoms from
atherosclerosis ...
Uses mainly relate to arterial thrombosis , and include : • acute myocardial
infarction high risk of myocardial infarction , including : – patients who have
recovered from myocardial infarction - patients with symptoms from
atherosclerosis ...
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Sommario
molecular aspects | 19 |
Method and measurement in pharmacology | 47 |
Absorption and distribution of drugs | 61 |
Copyright | |
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acetylcholine acid action activity agents agonists animals antagonists associated binding block blood body bone brain calcium cardiac cause cells central changes channels Chapter chemical clinical compounds concentration decrease dependence depression discussed disease dopamine dose drugs effects enzyme evidence example excretion factor function GABA gene given glucose glutamate growth half-life heart hormone human important increase inhibition inhibitors injection insulin interaction involved known less liver mainly mechanisms mediators membrane molecules myocardial infarction Nature nerve nervous system neurons noradrenaline normal occur oestrogens orally pain pathway patients peptides pharmacological phase plasma potential pressure prevent produce protein reactions receptors reduced regulation release response result risk role secretion selective shown similar smooth muscle specific stimulation structure studies substances synthesis Table terminals therapeutic therapy thyroid tissues transmission transmitter transport treat treatment trials types unwanted effects various vascular
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Informazioni bibliografiche
| Titolo | Pharmacology Churchill Livingstone |
| Autori | H. P. Rang, M. Maureen Dale, J. M. Ritter |
| Edizione | 4, illustrata, ristampa |
| Editore | Churchill Livingstone, 1999 |
| Provenienza dell'originale | la University of Michigan |
| Digitalizzato | 18 ago 2008 |
| ISBN | 0443059748, 9780443059742 |
| Lunghezza | 830 pagine |
| Esporta citazione | BiBTeX EndNote RefMan |