Cardiopulmonary Physical TherapyScot Irwin, Jan Stephen Tecklin Mosby, 1990 - 585 pagine |
Dall'interno del libro
Risultati 1-3 di 16
Pagina 31
... ( VO2 max ) . In turn , all variables that are affected by sympathetic nervous system activity , that is , heart rate , contractile state , and vasoconstriction , have also been shown to be linearly related to the fraction Vo2 / Vo2 In ...
... ( VO2 max ) . In turn , all variables that are affected by sympathetic nervous system activity , that is , heart rate , contractile state , and vasoconstriction , have also been shown to be linearly related to the fraction Vo2 / Vo2 In ...
Pagina 32
Scot Irwin, Jan Stephen Tecklin. max max . hemodynamic parameters plateau and ... Vo2 can be sustained for hours , whereas near- maximal exertion leads ... Vo2 Adjust- ments for minor variations in metabolic need are through two mechanisms ...
Scot Irwin, Jan Stephen Tecklin. max max . hemodynamic parameters plateau and ... Vo2 can be sustained for hours , whereas near- maximal exertion leads ... Vo2 Adjust- ments for minor variations in metabolic need are through two mechanisms ...
Pagina 515
... ( Vo2 max ) , oxygen extrac- tion ( a - vo2 ) , cardiac output , and stroke volume increase , whereas resting heart rate is found to decrease after train- ing . In general , oxygen consumption ( Vo2 ) and Vo2 are used to evaluate the ...
... ( Vo2 max ) , oxygen extrac- tion ( a - vo2 ) , cardiac output , and stroke volume increase , whereas resting heart rate is found to decrease after train- ing . In general , oxygen consumption ( Vo2 ) and Vo2 are used to evaluate the ...
Sommario
PART ONE Cardiac physical therapy PART TWO Pulmonary physical therapy | 1 |
Respiratory muscles 505 26 Evaluation and physical treatment of the patient with | 24 |
Respiratory rehabilitation of the patient with a spinal | 215 |
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Cardiopulmonary Physical Therapy, Volume 1 Scot Irwin,Jan Stephen Tecklin Visualizzazione estratti - 1985 |
Parole e frasi comuni
abnormal activity acute addition agents alveolar alveoli angina assessment associated blood pressure breathing bronchial capacity cardiac cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical common completed complications conditioning continue coronary artery decreased depression determined develop disease drainage drugs dysrhythmias early effects evaluation exercise test factors failure flow function heart rate hospital important improve increased indicated infant initial involved lead left ventricular less levels limited lower lung major maximal measured mechanical minutes monitoring muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed period persons phase physical therapy position present problems produce progression pulmonary reduced referred require resistance respiratory response Resting rhythm risk secretions severe significant signs sounds surgery symptoms systolic Table techniques therapist tion treatment usually values ventilation ventricular volume wall wave