Cardiopulmonary Physical TherapyScot Irwin, Jan Stephen Tecklin Mosby, 1990 - 585 pagine |
Dall'interno del libro
Risultati 1-3 di 76
Pagina 14
... cells , increased permeability , plate- let sticking , stimulation of smooth muscle cell proliferation " Trap - door " effect Destruction of endothelial cells Hypercontraction , swelling and loss of endothelial cells , platelet ...
... cells , increased permeability , plate- let sticking , stimulation of smooth muscle cell proliferation " Trap - door " effect Destruction of endothelial cells Hypercontraction , swelling and loss of endothelial cells , platelet ...
Pagina 41
... cells . The excitatory stimulus generated by the pacemaker cells within the SA node spreads to adjacent atrial myocardium and then , sequentially , to the AV node , His bundle , right and left bundle branches , distal Purkinje system ...
... cells . The excitatory stimulus generated by the pacemaker cells within the SA node spreads to adjacent atrial myocardium and then , sequentially , to the AV node , His bundle , right and left bundle branches , distal Purkinje system ...
Pagina 248
... cells . The pathological changes associated with chronic bron- chitis are found in the two sources of bronchial mucus- epithelial goblet cells and mucus - secreting bronchial glands . The glands produce almost all of the bronchial mu ...
... cells . The pathological changes associated with chronic bron- chitis are found in the two sources of bronchial mucus- epithelial goblet cells and mucus - secreting bronchial glands . The glands produce almost all of the bronchial mu ...
Sommario
PART ONE Cardiac physical therapy PART TWO Pulmonary physical therapy | 1 |
Respiratory muscles 505 26 Evaluation and physical treatment of the patient with | 24 |
Respiratory rehabilitation of the patient with a spinal | 215 |
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Cardiopulmonary Physical Therapy, Volume 1 Scot Irwin,Jan Stephen Tecklin Visualizzazione estratti - 1985 |
Parole e frasi comuni
abnormal activity acute addition agents alveolar alveoli angina assessment associated blood pressure breathing bronchial capacity cardiac cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical common completed complications conditioning continue coronary artery decreased depression determined develop disease drainage drugs dysrhythmias early effects evaluation exercise test factors failure flow function heart rate hospital important improve increased indicated infant initial involved lead left ventricular less levels limited lower lung major maximal measured mechanical minutes monitoring muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed period persons phase physical therapy position present problems produce progression pulmonary reduced referred require resistance respiratory response Resting rhythm risk secretions severe significant signs sounds surgery symptoms systolic Table techniques therapist tion treatment usually values ventilation ventricular volume wall wave