Cardiopulmonary Physical TherapyScot Irwin, Jan Stephen Tecklin Mosby, 1990 - 585 pagine |
Dall'interno del libro
Risultati 1-3 di 93
Pagina 27
... ventricular pressure is a function of instantaneous ventricular volume and is not affected by the volume at which ejection began . Loop A - E - F - I illustrates the effect of raising afterload ( aortic pressure ) on stroke volume ...
... ventricular pressure is a function of instantaneous ventricular volume and is not affected by the volume at which ejection began . Loop A - E - F - I illustrates the effect of raising afterload ( aortic pressure ) on stroke volume ...
Pagina 33
... function of heart rate , afterload , and contractile state and empirically ... ventricular volumes can be mea- sured and the percentage of blood filling ... ventricular dysfunction , there is an apparent absence of exercise - induced ...
... function of heart rate , afterload , and contractile state and empirically ... ventricular volumes can be mea- sured and the percentage of blood filling ... ventricular dysfunction , there is an apparent absence of exercise - induced ...
Pagina 160
Scot Irwin, Jan Stephen Tecklin. training in patients with poor left ventricular function with mild to moderate congestive heart failure also demon- strated the importance of regular aerobic exercise.23.148 Regular aerobic exercise that ...
Scot Irwin, Jan Stephen Tecklin. training in patients with poor left ventricular function with mild to moderate congestive heart failure also demon- strated the importance of regular aerobic exercise.23.148 Regular aerobic exercise that ...
Sommario
PART ONE Cardiac physical therapy PART TWO Pulmonary physical therapy | 1 |
Respiratory muscles 505 26 Evaluation and physical treatment of the patient with | 24 |
Respiratory rehabilitation of the patient with a spinal | 215 |
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Cardiopulmonary Physical Therapy, Volume 1 Scot Irwin,Jan Stephen Tecklin Visualizzazione estratti - 1985 |
Parole e frasi comuni
abnormal activity acute addition agents alveolar alveoli angina assessment associated blood pressure breathing bronchial capacity cardiac cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical common completed complications conditioning continue coronary artery decreased depression determined develop disease drainage drugs dysrhythmias early effects evaluation exercise test factors failure flow function heart rate hospital important improve increased indicated infant initial involved lead left ventricular less levels limited lower lung major maximal measured mechanical minutes monitoring muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed period persons phase physical therapy position present problems produce progression pulmonary reduced referred require resistance respiratory response Resting rhythm risk secretions severe significant signs sounds surgery symptoms systolic Table techniques therapist tion treatment usually values ventilation ventricular volume wall wave