Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 14
Pagina 29
... Vo2 / Vo2 The total peripheral resistance ( TPR ) is the net result of opposing vasoconstriction and vasodi- latation , and since the latter two ... max max exercise have gained major acceptance and support from Chapter 3 Hemodynamics 29.
... Vo2 / Vo2 The total peripheral resistance ( TPR ) is the net result of opposing vasoconstriction and vasodi- latation , and since the latter two ... max max exercise have gained major acceptance and support from Chapter 3 Hemodynamics 29.
Pagina 30
Scot Irwin, Jan Stephen Tecklin. max max exercise have gained major acceptance and support from the medical ... Vo2 compared to normals . In the cor- onary subject , as in the normal , the Vo2 depends on two main components , cardiac ...
Scot Irwin, Jan Stephen Tecklin. max max exercise have gained major acceptance and support from the medical ... Vo2 compared to normals . In the cor- onary subject , as in the normal , the Vo2 depends on two main components , cardiac ...
Pagina 31
... ( Vo2 ) . max . max Nevertheless , symptomatic improvement is seen . This occurs because at any given submaximal Vo2 , the relative exercise intensity after training is lower compared to pre- training performance ; that is , Vo2 / Vo22 is ...
... ( Vo2 ) . max . max Nevertheless , symptomatic improvement is seen . This occurs because at any given submaximal Vo2 , the relative exercise intensity after training is lower compared to pre- training performance ; that is , Vo2 / Vo22 is ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
Copyright | |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall