Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 18
Pagina 20
... Afterload Preload Fig . 3-2 . A model of fundamental muscle mechanics . Papillary muscle is affixed to a strain gauge and a movable lever that can be locked . Variations in loading sequence and locking the lever simu- lates the ...
... Afterload Preload Fig . 3-2 . A model of fundamental muscle mechanics . Papillary muscle is affixed to a strain gauge and a movable lever that can be locked . Variations in loading sequence and locking the lever simu- lates the ...
Pagina 22
... afterload . From equation ( 1 ) afterload , T , is a function of central aortic pressure , P , ventricular size , R , and wall thickness , h . If we assume a constant wall thickness and integrate instantaneous afterload throughout all ...
... afterload . From equation ( 1 ) afterload , T , is a function of central aortic pressure , P , ventricular size , R , and wall thickness , h . If we assume a constant wall thickness and integrate instantaneous afterload throughout all ...
Pagina 23
... afterload . As shown in later sections , the converse is true as well ; that is , an increase in stroke volume can be affected by afterload reduction . Fig . 3-7 , B , illustrates the effect of adding an inotropic agent to the intact ...
... afterload . As shown in later sections , the converse is true as well ; that is , an increase in stroke volume can be affected by afterload reduction . Fig . 3-7 , B , illustrates the effect of adding an inotropic agent to the intact ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
Copyright | |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall