Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 9
Pagina 37
... beta block- ade interferes with catecholamine - mediated platelet aggre ... blockade ) at low dosage levels , but this advantage is lost at higher dos- ages ... blockers : 1. Severe sinus bradycardia , second and third degree AV block ...
... beta block- ade interferes with catecholamine - mediated platelet aggre ... blockade ) at low dosage levels , but this advantage is lost at higher dos- ages ... blockers : 1. Severe sinus bradycardia , second and third degree AV block ...
Pagina 47
... beta ad- renergic blocking agent and a potent diuretic . Minoxidil ( Loniten ) ... blockers exhibit moderate antihypertensive ac- tivity when employed alone ... Beta blockers are often prescribed along with vasodilators to augment the ...
... beta ad- renergic blocking agent and a potent diuretic . Minoxidil ( Loniten ) ... blockers exhibit moderate antihypertensive ac- tivity when employed alone ... Beta blockers are often prescribed along with vasodilators to augment the ...
Pagina 48
... beta blockers , and calcium channel blockers . Nitrates reviewed include nitroglycerin , isosorbide di- nitrate ( Isordil , Sorbitrate ) , and topical nitroglycerin ( Ni- trobid , Nitrol , Transderm - Nitro , and Nitrodur ) . Beta ...
... beta blockers , and calcium channel blockers . Nitrates reviewed include nitroglycerin , isosorbide di- nitrate ( Isordil , Sorbitrate ) , and topical nitroglycerin ( Ni- trobid , Nitrol , Transderm - Nitro , and Nitrodur ) . Beta ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall