Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
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Pagina 12
... cells Elastic fibers Fig . 2-7 . Enlargement of a section of a coronary artery and its structural components , namely , the endothelium , the media con- sisting primarily of smooth muscle cells , and the adventitia . ( From Benditt ...
... cells Elastic fibers Fig . 2-7 . Enlargement of a section of a coronary artery and its structural components , namely , the endothelium , the media con- sisting primarily of smooth muscle cells , and the adventitia . ( From Benditt ...
Pagina 38
... cells in the sino- atrial and atrioventricular nodes and contributes to the pla- teau phase ( phase 2 ) of ordinary myocardium and the spe- cialized cells of the His - Purkinje system . Predictably , the calcium - blocking drugs may ...
... cells in the sino- atrial and atrioventricular nodes and contributes to the pla- teau phase ( phase 2 ) of ordinary myocardium and the spe- cialized cells of the His - Purkinje system . Predictably , the calcium - blocking drugs may ...
Pagina 187
... cells results in the production of carbon dioxide , which diffuses out of the cells into the tissue capillaries and is carried by the blood to the lungs , where it diffuses into the alveolar air and is expired . As oxygen diffuses into ...
... cells results in the production of carbon dioxide , which diffuses out of the cells into the tissue capillaries and is carried by the blood to the lungs , where it diffuses into the alveolar air and is expired . As oxygen diffuses into ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall