Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 60
Pagina 181
... Chest - wall compliance . Like the lung , the chest wall is elastic . If air is introduced into the pleural cavity , the lungs will collapse inward and the chest wall will expand outward ( Fig . 10-17 ) . As previously discussed , there ...
... Chest - wall compliance . Like the lung , the chest wall is elastic . If air is introduced into the pleural cavity , the lungs will collapse inward and the chest wall will expand outward ( Fig . 10-17 ) . As previously discussed , there ...
Pagina 218
... Chest wall Upper abdominal viscera Aching | Deep pressure Generally localized C5 - T1 Chest wall ; apical parietal pleura T2 - T8 Dull Remainder parietal ; upper pericardial Deep Ischemia pleura Thoracic Aching Distention T6 - T8 ...
... Chest wall Upper abdominal viscera Aching | Deep pressure Generally localized C5 - T1 Chest wall ; apical parietal pleura T2 - T8 Dull Remainder parietal ; upper pericardial Deep Ischemia pleura Thoracic Aching Distention T6 - T8 ...
Pagina 297
... chest wall during a deep breath.5.6 The anterior - apical and lateral - basilar areas of the chest wall usually move freely with deep inspiratory efforts . The therapist's hands are placed over these areas , unilaterally or bilaterally ...
... chest wall during a deep breath.5.6 The anterior - apical and lateral - basilar areas of the chest wall usually move freely with deep inspiratory efforts . The therapist's hands are placed over these areas , unilaterally or bilaterally ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
Copyright | |
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Parole e frasi comuni
abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall