Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 10
Pagina 39
... digoxin is given alone . Thus the dosage of digoxin must be dimin- ished when quinidine is added to the therapeutic regimen . Because of their extensive electrophysiological activity , quinidine , procaine amide , and disopyramide are ...
... digoxin is given alone . Thus the dosage of digoxin must be dimin- ished when quinidine is added to the therapeutic regimen . Because of their extensive electrophysiological activity , quinidine , procaine amide , and disopyramide are ...
Pagina 40
... digoxin concentration , and digitalis in- toxication in patients receiving digoxin therapy 5. Cinchonism ; tinnitus , headache , nausea , visual distur- bances 6. Nausea , vomiting , abdominal pain , diarrhea 7. Hemolytic anemia ...
... digoxin concentration , and digitalis in- toxication in patients receiving digoxin therapy 5. Cinchonism ; tinnitus , headache , nausea , visual distur- bances 6. Nausea , vomiting , abdominal pain , diarrhea 7. Hemolytic anemia ...
Pagina 43
... Digoxin Lanoxin Lanotoside C Deslanoside Digoxin USP Cedilanid Cedilanid D Oral and par- enteral 36 hours 3 to 6 days Renal Oral 36 hours 3 to 6 days Parenteral 33 hours 3 to 6 days Renal Renal Digitalis leaf Various manu- Oral 40 % G ...
... Digoxin Lanoxin Lanotoside C Deslanoside Digoxin USP Cedilanid Cedilanid D Oral and par- enteral 36 hours 3 to 6 days Renal Oral 36 hours 3 to 6 days Parenteral 33 hours 3 to 6 days Renal Renal Digitalis leaf Various manu- Oral 40 % G ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall