Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 39
Pagina 36
... Hypotension is usually asso- ciated with sympathetically mediated reflex tachycardia , which , if severe , may aggravate myocardial ischemia . Al- though hypotension diminishes myocardial work , and therefore reduces MVO2 , severe ...
... Hypotension is usually asso- ciated with sympathetically mediated reflex tachycardia , which , if severe , may aggravate myocardial ischemia . Al- though hypotension diminishes myocardial work , and therefore reduces MVO2 , severe ...
Pagina 40
... hypotension , in patients with borderline cardiac function or overt congestive heart failure 3. Severe bradycardia , AV block , asystole ; careful monitor- ing required in patients with sinus node dysfunction and preexisting conduction ...
... hypotension , in patients with borderline cardiac function or overt congestive heart failure 3. Severe bradycardia , AV block , asystole ; careful monitor- ing required in patients with sinus node dysfunction and preexisting conduction ...
Pagina 90
... Hypotension ( PB , 80/50 ) Hypertension ( BP , 170/100 ) at rest Uncontrolled arrhythmias before exercise such as atrial fi- brillation , frequent , or complex premature ventricular con- tractions Table 6-8 . End points for low - level ...
... Hypotension ( PB , 80/50 ) Hypertension ( BP , 170/100 ) at rest Uncontrolled arrhythmias before exercise such as atrial fi- brillation , frequent , or complex premature ventricular con- tractions Table 6-8 . End points for low - level ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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Parole e frasi comuni
abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall