Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 94
Pagina 23
... Increased preload , G - H - C - D . Increased afterload , A - E - F - I . Increased contractile state , A - B - J - K . Pharmacological considerations MVO , demand > supply Electrocardiographic abnormalities Fig. B1 A B A Volume D1 ...
... Increased preload , G - H - C - D . Increased afterload , A - E - F - I . Increased contractile state , A - B - J - K . Pharmacological considerations MVO , demand > supply Electrocardiographic abnormalities Fig. B1 A B A Volume D1 ...
Pagina 36
... ( increased heart rate ) and augmented myocardial contractility ( positive inotropic effect ) , two major deter- minants of MVO2 . Accordingly , the increased sympathetic activity that regularly accompanies ordinary physical and emotional ...
... ( increased heart rate ) and augmented myocardial contractility ( positive inotropic effect ) , two major deter- minants of MVO2 . Accordingly , the increased sympathetic activity that regularly accompanies ordinary physical and emotional ...
Pagina 198
... increased work of breathing to maintain ventilation . Inability to meet this increased demand for muscular work may lead to CO2 re- tention , right - sided congestive heart failure , and cor pul- monale . Little is known , however ...
... increased work of breathing to maintain ventilation . Inability to meet this increased demand for muscular work may lead to CO2 re- tention , right - sided congestive heart failure , and cor pul- monale . Little is known , however ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall