Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 66
Pagina 34
... ischemia . Sinus bradycardia first- , second- , third - degree AV. Determinants of MVO , Major Heart rate Contractile state Wall tension Systolic pressure Ventricular volume Minor Basal O , requirements Activation energy Fiber shortening ...
... ischemia . Sinus bradycardia first- , second- , third - degree AV. Determinants of MVO , Major Heart rate Contractile state Wall tension Systolic pressure Ventricular volume Minor Basal O , requirements Activation energy Fiber shortening ...
Pagina 91
... ischemia are not clearly understood . Ellestad has proposed that the ischemic electrophysiological changes result from both hemody- namic and metabolic factors including elevated left ven- tricular end - diastolic pressures , incomplete ...
... ischemia are not clearly understood . Ellestad has proposed that the ischemic electrophysiological changes result from both hemody- namic and metabolic factors including elevated left ven- tricular end - diastolic pressures , incomplete ...
Pagina 92
... ischemia is not 100 % and , in fact , is related directly to the prevalence of the disease in the population of patients being tested ( Table 6- 10 ) . In other words , the predictive value of exercise - in- duced ST - segment changes ...
... ischemia is not 100 % and , in fact , is related directly to the prevalence of the disease in the population of patients being tested ( Table 6- 10 ) . In other words , the predictive value of exercise - in- duced ST - segment changes ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall