Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 82
Pagina 177
... divided by the total number of breaths ( n ) and multiplied by the number of breaths per minute ( f ) . Therefore : VOLUME Normal Spirogram Obstructive Lung Disease W FEV3.0 -FEF 25-757 Chapter 10 Respiratory physiology 177.
... divided by the total number of breaths ( n ) and multiplied by the number of breaths per minute ( f ) . Therefore : VOLUME Normal Spirogram Obstructive Lung Disease W FEV3.0 -FEF 25-757 Chapter 10 Respiratory physiology 177.
Pagina 231
... lung disease Decreased arterial oxygen tension in bilateral lung disease Decreased arterial oxygen tension lying on the affected lung in unilateral lung disease Decreased arterial oxygen tension lying on the left side in bilateral lung ...
... lung disease Decreased arterial oxygen tension in bilateral lung disease Decreased arterial oxygen tension lying on the affected lung in unilateral lung disease Decreased arterial oxygen tension lying on the left side in bilateral lung ...
Pagina 340
... lung disease . As the pulmonary involve- ment progresses , habitual coughing , abnormal distribution of ventilation , decreased pulmonary function , and weight loss compromise exercise tolerance . Complications of the pulmonary disease ...
... lung disease . As the pulmonary involve- ment progresses , habitual coughing , abnormal distribution of ventilation , decreased pulmonary function , and weight loss compromise exercise tolerance . Complications of the pulmonary disease ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall