Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 76
Pagina 175
... lung vol- ume . Similarly , dead space increases as a function of body height , bronchodilator drugs , diseases such ... volume changes tend to be similar . However , at low lung volumes , airways in the dependent portion close , and ...
... lung vol- ume . Similarly , dead space increases as a function of body height , bronchodilator drugs , diseases such ... volume changes tend to be similar . However , at low lung volumes , airways in the dependent portion close , and ...
Pagina 180
... tidal volume stays in the conducting airways ( anatomic dead space ) . Nitrogen concentration begins to rise as the airways are cleared and alveolar gas is expired . The recorded volume to this point represents anatomical dead space ...
... tidal volume stays in the conducting airways ( anatomic dead space ) . Nitrogen concentration begins to rise as the airways are cleared and alveolar gas is expired . The recorded volume to this point represents anatomical dead space ...
Pagina 384
Scot Irwin, Jan Stephen Tecklin. Respiratory muscle strength The strength of the respiratory muscle contraction is di ... lung volume . 12,16 Inspiratory and expiratory muscle strength are deter- mined by measurement of the maximum static ...
Scot Irwin, Jan Stephen Tecklin. Respiratory muscle strength The strength of the respiratory muscle contraction is di ... lung volume . 12,16 Inspiratory and expiratory muscle strength are deter- mined by measurement of the maximum static ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall