Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 78
Pagina 15
... myocardial oxygen demand and coronary flow is discussed in Chapter 4 . ACUTE MYOCARDIAL INFARCTION Prolonged ischemia as a result of complete occlusion of a coronary artery or a severe occlusion coupled with in- creased vasomotor tone ...
... myocardial oxygen demand and coronary flow is discussed in Chapter 4 . ACUTE MYOCARDIAL INFARCTION Prolonged ischemia as a result of complete occlusion of a coronary artery or a severe occlusion coupled with in- creased vasomotor tone ...
Pagina 134
... myocardial ischemia , low - potassium or low - magne- sium levels within the myocardium , inadequate intake of linoleic acid in the diet , and so on . In any case , we can certainly say that this man experienced a greatly improved ...
... myocardial ischemia , low - potassium or low - magne- sium levels within the myocardium , inadequate intake of linoleic acid in the diet , and so on . In any case , we can certainly say that this man experienced a greatly improved ...
Pagina 135
... myocardial ischemia , low - potassium or low - magne- sium levels within the myocardium , inadequate intake of linoleic acid in the diet , and so on . In any case , we can certainly say that this man experienced a greatly improved ...
... myocardial ischemia , low - potassium or low - magne- sium levels within the myocardium , inadequate intake of linoleic acid in the diet , and so on . In any case , we can certainly say that this man experienced a greatly improved ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall