Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 14
Pagina 8
... node artery , and in approximately 10 % of the cases , the posterior descending artery off the cir- cumflex ( via a posterior septal branch ) supplies the AV node . Right coronary artery system The right coronary artery ( RCA ) ...
... node artery , and in approximately 10 % of the cases , the posterior descending artery off the cir- cumflex ( via a posterior septal branch ) supplies the AV node . Right coronary artery system The right coronary artery ( RCA ) ...
Pagina 38
... nodes . Verapamil , on the other hand , has been associated with bradycardia secondary to sinus node slowing or AV node conduction delay , or both . Because of their negative ino- tropic activity , both agents may aggravate congestive ...
... nodes . Verapamil , on the other hand , has been associated with bradycardia secondary to sinus node slowing or AV node conduction delay , or both . Because of their negative ino- tropic activity , both agents may aggravate congestive ...
Pagina 43
... node AV node Purkinje Excitability Atrium Ventricle Purkinje Effective refractory period Atrium Ventricle AV node Purkinje Conduction velocity Atrium Ventricle AV node Purkinje D , Decreased ; / , increased ; no change . - Effect with ...
... node AV node Purkinje Excitability Atrium Ventricle Purkinje Effective refractory period Atrium Ventricle AV node Purkinje Conduction velocity Atrium Ventricle AV node Purkinje D , Decreased ; / , increased ; no change . - Effect with ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall