Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
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Pagina 94
... onset is the more powerful prognostic variable . The onset of an- gina early in the test ( within the first 3 minutes ) , in com- bination with early onset of ST depression , worsens the prognosis for the patient as well.11 The ...
... onset is the more powerful prognostic variable . The onset of an- gina early in the test ( within the first 3 minutes ) , in com- bination with early onset of ST depression , worsens the prognosis for the patient as well.11 The ...
Pagina 121
... onset of angina : 14 × 103 RPP at onset of ST - segment changes : ( not measured ) 1.5 mm ST - segment depression at maximal RPP No arrhythmias Fair symptom - limited physical work capacity The cardiologist subsequently started the ...
... onset of angina : 14 × 103 RPP at onset of ST - segment changes : ( not measured ) 1.5 mm ST - segment depression at maximal RPP No arrhythmias Fair symptom - limited physical work capacity The cardiologist subsequently started the ...
Pagina 121
... onset of angina : 14 × 103 RPP at onset of ST - segment changes : ( not measured ) 1.5 mm ST - segment depression at maximal RPP No arrhythmias Fair symptom - limited physical work capacity The cardiologist subsequently started the ...
... onset of angina : 14 × 103 RPP at onset of ST - segment changes : ( not measured ) 1.5 mm ST - segment depression at maximal RPP No arrhythmias Fair symptom - limited physical work capacity The cardiologist subsequently started the ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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Parole e frasi comuni
abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall