Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 60
Pagina 27
... peripheral veins centrally to the right atrium . Venoconstriction alone by raising peripheral ve- nous pressure can roughly double venous return . In Fig . 3-12 , venous return is plotted against right atrial pressure , the graphic ...
... peripheral veins centrally to the right atrium . Venoconstriction alone by raising peripheral ve- nous pressure can roughly double venous return . In Fig . 3-12 , venous return is plotted against right atrial pressure , the graphic ...
Pagina 28
... peripheral adjustments . Point B ( Fig . 3-13 ) is reached when the maximal periph- eral vasoconstriction is achieved , with the elevated venous tone enhancing venous return about twofold . At this point , it is important to recognize ...
... peripheral adjustments . Point B ( Fig . 3-13 ) is reached when the maximal periph- eral vasoconstriction is achieved , with the elevated venous tone enhancing venous return about twofold . At this point , it is important to recognize ...
Pagina 194
... peripheral chemoreceptors , peripheral reflexes from the lungs and other parts of the body , and by the cortical centers . Central and peripheral chemoreceptor mechanisms The dominant regulation of the respiratory centers usu- ally ...
... peripheral chemoreceptors , peripheral reflexes from the lungs and other parts of the body , and by the cortical centers . Central and peripheral chemoreceptor mechanisms The dominant regulation of the respiratory centers usu- ally ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall