Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 19
Pagina 74
... premature ventricular contractions ( PVC's ) . Common character- istics of PVC's are that ( 1 ) they are early , ( 2 ) they have no conducted P wave , ( 3 ) they have a wide bizarre QRST , and ( 4 ) there is a complete compensatory ...
... premature ventricular contractions ( PVC's ) . Common character- istics of PVC's are that ( 1 ) they are early , ( 2 ) they have no conducted P wave , ( 3 ) they have a wide bizarre QRST , and ( 4 ) there is a complete compensatory ...
Pagina 102
... premature ventricular contractions becoming mul- tifocal . 4. Recurrent coupled premature ventricular contractions or ven- tricular tachycardia ( three PVC's in a row ) . 5. Development of rapid atrial arrhythmias . 6. Level III angina ...
... premature ventricular contractions becoming mul- tifocal . 4. Recurrent coupled premature ventricular contractions or ven- tricular tachycardia ( three PVC's in a row ) . 5. Development of rapid atrial arrhythmias . 6. Level III angina ...
Pagina 125
... premature atrial and ventricular contractions being noticed . Jan. 17 , 1981 : Patient noticed the onset of slight precordial chest pain lasting approximately 30 minutes and subsiding with- out medications . Repeat electrocardiogram ...
... premature atrial and ventricular contractions being noticed . Jan. 17 , 1981 : Patient noticed the onset of slight precordial chest pain lasting approximately 30 minutes and subsiding with- out medications . Repeat electrocardiogram ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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Parole e frasi comuni
abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall