Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
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Pagina 54
... Systolic 190- blood 180- pressure ( mm Hg ) 170- 160- 150- 140- 130- Rest- 90- Diastolic pressure 80- 70- Increasing exercise work load Fig . 5-8 . Normal systolic and diastolic blood responses to exertion . scribed as a heart rate ...
... Systolic 190- blood 180- pressure ( mm Hg ) 170- 160- 150- 140- 130- Rest- 90- Diastolic pressure 80- 70- Increasing exercise work load Fig . 5-8 . Normal systolic and diastolic blood responses to exertion . scribed as a heart rate ...
Pagina 55
Scot Irwin, Jan Stephen Tecklin. 170- 160 170 Systolic blood pressure 150- 140- 130 120- 110- 100- Rest Increasing levels of exercise Fig . 5-10 . Abnormal systolic blood pressure response to exertion . Striking fall in systolic pressure ...
Scot Irwin, Jan Stephen Tecklin. 170- 160 170 Systolic blood pressure 150- 140- 130 120- 110- 100- Rest Increasing levels of exercise Fig . 5-10 . Abnormal systolic blood pressure response to exertion . Striking fall in systolic pressure ...
Pagina 56
... systolic blood pressure to prolonged exercise at the same work load . occurs with an increase in work load . In Fig . 5-11 the work loads were increased at minutes 3 and 6. Systolic pressure fell 20 mm Hg or more with an increase in ...
... systolic blood pressure to prolonged exercise at the same work load . occurs with an increase in work load . In Fig . 5-11 the work loads were increased at minutes 3 and 6. Systolic pressure fell 20 mm Hg or more with an increase in ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall