Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 66
Pagina 57
... tients with angina are unable to exceed their anginal rate pressure product.8.28 An example of this is given below ... tients who actually eliminate their angina completely . Pa- tients who are capable of increasing or eliminating their ...
... tients with angina are unable to exceed their anginal rate pressure product.8.28 An example of this is given below ... tients who actually eliminate their angina completely . Pa- tients who are capable of increasing or eliminating their ...
Pagina 138
... tients also demonstrated increased total cholesterol values ( p < .05 ) as well . 59 Both Oschrin and Erkelens24 documented increased HDL - C values after 6 months ' training of 11 % to 14 % above pretraining values . The major ...
... tients also demonstrated increased total cholesterol values ( p < .05 ) as well . 59 Both Oschrin and Erkelens24 documented increased HDL - C values after 6 months ' training of 11 % to 14 % above pretraining values . The major ...
Pagina 227
... tients with lung disease , however , limited exercise toler- ance may be the result of impaired oxygen transport , im- paired pulmonary circulation , metabolic disturbances or disturbances of respiratory regulation , and the sensation ...
... tients with lung disease , however , limited exercise toler- ance may be the result of impaired oxygen transport , im- paired pulmonary circulation , metabolic disturbances or disturbances of respiratory regulation , and the sensation ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
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Parole e frasi comuni
abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall