Cardiopulmonary Physical Therapy, Volume 1Scot Irwin, Jan Stephen Tecklin Mosby, 1985 - 473 pagine |
Dall'interno del libro
Risultati 1-3 di 4
Pagina 29
... workload , TPR too varies in an inverse linear re- lation to VO2 / VO2 max . Within seconds of onset of exercise , the heart rate be- gins to rise and may double within 15 seconds . Because it is easily measured , the heart rate ...
... workload , TPR too varies in an inverse linear re- lation to VO2 / VO2 max . Within seconds of onset of exercise , the heart rate be- gins to rise and may double within 15 seconds . Because it is easily measured , the heart rate ...
Pagina 99
... Workload Time HR Supine. APPENDIX A Guidelines for maximal exercise testing Definition A maximal symptom - limited exercise test is an electrocardi- ographically monitored evaluation of a person's maximal oxy- gen consumption during ...
... Workload Time HR Supine. APPENDIX A Guidelines for maximal exercise testing Definition A maximal symptom - limited exercise test is an electrocardi- ographically monitored evaluation of a person's maximal oxy- gen consumption during ...
Pagina 100
Scot Irwin, Jan Stephen Tecklin. TREADMILL TEST WORKSHEET - cont'd STAGE Workload Time HR Supine Pre - Exercise Supine Pre - Exercise 2 Breath - Holding 3 Hyperventilation 3 Sitting Pre - Exercise 4 Standing Pre - Exercise 5 1 2 3 4 5 6 ...
Scot Irwin, Jan Stephen Tecklin. TREADMILL TEST WORKSHEET - cont'd STAGE Workload Time HR Supine Pre - Exercise Supine Pre - Exercise 2 Breath - Holding 3 Hyperventilation 3 Sitting Pre - Exercise 4 Standing Pre - Exercise 5 1 2 3 4 5 6 ...
Sommario
an overview of the basic mechanism | 6 |
Physical therapy for the child with respiratory 21 Respiratory rehabilitation of the patient with a spinal | 18 |
Hemodynamics | 19 |
Copyright | |
7 sezioni non visualizzate
Altre edizioni - Visualizza tutto
Parole e frasi comuni
abnormal activity acute addition airway alveolar alveoli angina arrhythmias assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cause cells changes Chapter chest chronic Circulation clinical complete complications continued contraction coronary artery disease decrease depression determined develop discussed disease drainage drugs effects evaluation exercise testing factors failure flow force function further heart rate hypertension important improve increased indicate initial inspiration intensity involved less limited load lower lung major maximal measured mechanical minutes monitored muscle myocardial infarction normal obstructive occur oxygen pain patient peak performed peripheral phase physical therapy position prevent produce progression pulmonary reduced resistance respiratory response Resting rise risk secretions segment severe significant signs sounds surgery symptoms systolic Table therapist thoracotomy tients tion tissue tolerance treatment usually vascular venous ventilation ventricular volume wall