Cardiopulmonary Physical TherapyScot Irwin, Jan Stephen Tecklin Mosby, 1995 - 620 pagine |
Dall'interno del libro
Risultati 1-3 di 83
Pagina 13
... cells , increased permeability , platelet sticking , stimulation of smooth muscle cell proliferation " Trap - door " effect Destruction of endothelial cells Hypercontraction , swelling and loss . of endothelial cells , platelet ...
... cells , increased permeability , platelet sticking , stimulation of smooth muscle cell proliferation " Trap - door " effect Destruction of endothelial cells Hypercontraction , swelling and loss . of endothelial cells , platelet ...
Pagina 42
... cells . The excitatory stimulus generated by the pacemaker cells within . the SA node spreads to adjacent atrial myocardium and then , sequentially , to the AV node , His bundle , right and left bundle branches , distal Purkinje system ...
... cells . The excitatory stimulus generated by the pacemaker cells within . the SA node spreads to adjacent atrial myocardium and then , sequentially , to the AV node , His bundle , right and left bundle branches , distal Purkinje system ...
Pagina 268
... cells . The pathological changes associated with chronic bron- chitis are found in the two sources of bronchial mucus- epithelial goblet cells and mucous - secreting bronchial glands . The glands produce almost all of the bronchial ...
... cells . The pathological changes associated with chronic bron- chitis are found in the two sources of bronchial mucus- epithelial goblet cells and mucous - secreting bronchial glands . The glands produce almost all of the bronchial ...
Sommario
PART ONE PART | 1 |
Physical Rehabilitation of the 27 Exercise Testing and Exercise Conditioning | 24 |
Physical Therapy for the Child with Respiratory | 235 |
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Parole e frasi comuni
abnormal activity acute addition agents alveolar alveoli angina assessment associated blood pressure breathing capacity cardiac output cardiac rehabilitation cardiovascular cause cells changes Chapter chest chronic Circulation clinical common completed complications continued coronary artery disease decreased demonstrated depression disease drugs dysrhythmias early effects evaluation exercise testing exercise training factors failure flow function heart rate hypertension important improve increased indicated initial intensity lead left ventricular less levels limited lower lung major maximal maximum measured mechanisms minutes monitoring muscle myocardial infarction normal obstructive occur oxygen pain patients peak performed period peripheral persons phase physical therapy position present produce progression pulmonary reduced reported require respiratory response rest rhythm risk severe significant specific surgery symptoms systolic therapist thoracotomy tion tissue treatment usually values vascular venous ventilation ventricular volume wall wave workload