PharmacologyChurchill Livingstone, 2001 - 839 pagine Adapted for the U.S., this New Edition of PHARMACOLOGY, a popular UK text, describes what drugs do and emphasizes the mechanisms by which they act. The U.S. edition is revised and updated to reflect the substantial differences in drug names, FDA approvals, terminology, and guidelines for use. It also boasts new and revised chapters to ensure that students are up to date on the very latest information. Each chapter begins with a summary of information essential to the chapter and key points are highlighted within the text to alert students to crucial concepts. Spanish version also available, ISBN: 84-8174-492-1 |
Dall'interno del libro
Risultati 1-3 di 87
Pagina 97
... nerve as filaments that run to the paravertebral chain of sympathetic ganglia , which lies bilaterally on either side of the spinal column . Each preganglionic nerve branches and makes synaptic contact with ganglion cells in several ...
... nerve as filaments that run to the paravertebral chain of sympathetic ganglia , which lies bilaterally on either side of the spinal column . Each preganglionic nerve branches and makes synaptic contact with ganglion cells in several ...
Pagina 144
... nerve terminal . ATP itself has a transmitter function at adrenergic synapses ( see Lundberg 1996 ) being responsible for the fast excitatory synaptic potential and the rapid phase of contraction produced by sympathetic nerve activity ...
... nerve terminal . ATP itself has a transmitter function at adrenergic synapses ( see Lundberg 1996 ) being responsible for the fast excitatory synaptic potential and the rapid phase of contraction produced by sympathetic nerve activity ...
Pagina 157
H. P. Rang. By preventing exocytosis from occurring in response to depolarization of the nerve terminal ( noradrenergic neuron - blocking drugs ) . • By evoking norepinephrine release in the absence of nerve terminal depolarization ...
H. P. Rang. By preventing exocytosis from occurring in response to depolarization of the nerve terminal ( noradrenergic neuron - blocking drugs ) . • By evoking norepinephrine release in the absence of nerve terminal depolarization ...
Sommario
general principles | 11 |
molecular aspects | 19 |
Method and measurement in pharmacology | 47 |
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5-HT receptors absorption acetylcholine action potential activity adrenoceptors agents agonists amino acids anesthetics antidepressant antipsychotic asthma benzodiazepines binding block blood bradykinin brain Ca2+ cancer cardiac cause cells chemical cholinergic clinical compounds cytokines decrease depolarization depression disease diuretics dopamine dose drugs enzyme estrogens ethanol excretion factors function GABA gastrointestinal gene glucocorticoids glucose glutamate histamine hormone important inactivation increase inflammatory inhibition inhibitors injection insulin interaction intracellular intravenous kinase leptin liver mainly mechanism of action mediators membrane metabolism metabolites molecular molecules morphine muscarinic nerve terminals neurons nicotine norepinephrine normal occurs opioid orally oxide pathway patients peptide peripheral Pharmacokinetic Pharmacol pharmacological physiological pituitary plasma concentration platelet presynaptic produce prostaglandins protein reactions receptor antagonists reduced release renal response result role secretion side-effects smooth muscle sodium stimulation substance sympathetic synaptic synthesis therapeutic therapy tissue toxicity transmission transmitter treatment tubule unwanted effects uptake vascular vasoconstriction vitamin