PharmacologyChurchill Livingstone, 2001 - 839 pagine Adapted for the U.S., this New Edition of PHARMACOLOGY, a popular UK text, describes what drugs do and emphasizes the mechanisms by which they act. The U.S. edition is revised and updated to reflect the substantial differences in drug names, FDA approvals, terminology, and guidelines for use. It also boasts new and revised chapters to ensure that students are up to date on the very latest information. Each chapter begins with a summary of information essential to the chapter and key points are highlighted within the text to alert students to crucial concepts. Spanish version also available, ISBN: 84-8174-492-1 |
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Risultati 1-3 di 81
Pagina 33
... protein ( Target 1 , which may be an enzyme such as adenylate cyclase , or an ion channel ) . The By complex may also activate a target protein ( Target 2 ) . The GTPase activity of the a - subunit is increased when the target protein ...
... protein ( Target 1 , which may be an enzyme such as adenylate cyclase , or an ion channel ) . The By complex may also activate a target protein ( Target 2 ) . The GTPase activity of the a - subunit is increased when the target protein ...
Pagina 34
... protein family * These variants give rise to three main classes of G - protein ( G , G , and G ) , which show selectivity with respect to both the receptors and the effectors with which they couple , having specific recognition domains ...
... protein family * These variants give rise to three main classes of G - protein ( G , G , and G ) , which show selectivity with respect to both the receptors and the effectors with which they couple , having specific recognition domains ...
Pagina 39
... protein kinase , protein kinase C ( PKC ) , which causes phosphorylation of serine and threonine residues of a variety of intracellular proteins ( see Nishizuka 1986 , Parker & Dekker 1996 , Walaas & Greengard 1991 ) . DAG , unlike the ...
... protein kinase , protein kinase C ( PKC ) , which causes phosphorylation of serine and threonine residues of a variety of intracellular proteins ( see Nishizuka 1986 , Parker & Dekker 1996 , Walaas & Greengard 1991 ) . DAG , unlike the ...
Sommario
general principles | 11 |
molecular aspects | 19 |
Method and measurement in pharmacology | 47 |
Copyright | |
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5-HT receptors absorption acetylcholine action potential activity adrenoceptors agents agonists amino acids anesthetics antidepressant antipsychotic asthma benzodiazepines binding block blood bradykinin brain Ca2+ cancer cardiac cause cells chemical cholinergic clinical compounds cytokines decrease depolarization depression disease diuretics dopamine dose drugs enzyme estrogens ethanol excretion factors function GABA gastrointestinal gene glucocorticoids glucose glutamate histamine hormone important inactivation increase inflammatory inhibition inhibitors injection insulin interaction intracellular intravenous kinase leptin liver mainly mechanism of action mediators membrane metabolism metabolites molecular molecules morphine muscarinic nerve terminals neurons nicotine norepinephrine normal occurs opioid orally oxide pathway patients peptide peripheral Pharmacokinetic Pharmacol pharmacological physiological pituitary plasma concentration platelet presynaptic produce prostaglandins protein reactions receptor antagonists reduced release renal response result role secretion side-effects smooth muscle sodium stimulation substance sympathetic synaptic synthesis therapeutic therapy tissue toxicity transmission transmitter treatment tubule unwanted effects uptake vascular vasoconstriction vitamin