PharmacologyAdapted for the U.S., this New Edition of PHARMACOLOGY, a popular UK text, describes what drugs do and emphasizes the mechanisms by which they act. The U.S. edition is revised and updated to reflect the substantial differences in drug names, FDA approvals, terminology, and guidelines for use. It also boasts new and revised chapters to ensure that students are up to date on the very latest information. Each chapter begins with a summary of information essential to the chapter and key points are highlighted within the text to alert students to crucial concepts. Spanish version also available, ISBN: 84-8174-492-1 |
Dall'interno del libro
Risultati 1-3 di 88
Pagina 85
Table 5.4 Some drugs and related actively secreted Into the proximal renal
substances that are Acids Acetazolamide p-Aminohippuric acid Aminosalicylic
acid Cefaloridine Furosemide Glucuronic acid conjugates Glycine conjugates ...
Table 5.4 Some drugs and related actively secreted Into the proximal renal
substances that are Acids Acetazolamide p-Aminohippuric acid Aminosalicylic
acid Cefaloridine Furosemide Glucuronic acid conjugates Glycine conjugates ...
Pagina 300
828-834 (A synthetic analog of atrial natriuretic peptide may have improved
survival in those patients with oliguria but worsened it in those with non-oliguric
renal failure) Brindley G S 1986 Pilot experiments on the actions of drugs injected
into ...
828-834 (A synthetic analog of atrial natriuretic peptide may have improved
survival in those patients with oliguria but worsened it in those with non-oliguric
renal failure) Brindley G S 1986 Pilot experiments on the actions of drugs injected
into ...
Pagina 28
Cyclosporine, used to prevent transplant rejection (see Ch. 13), causes renal
damage via a change in renal vascular dynamics: a persistent increase in renal
vascular resistance with a marked decline in glomerular filtration rate and
systemic ...
Cyclosporine, used to prevent transplant rejection (see Ch. 13), causes renal
damage via a change in renal vascular dynamics: a persistent increase in renal
vascular resistance with a marked decline in glomerular filtration rate and
systemic ...
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Sommario
general principles | 2 |
molecular aspects | 19 |
Method and measurement in pharmacology | 47 |
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absorption acetylcholine acid action activity agents agonists anesthetic antagonists associated binding block blood body brain calcium cardiac cause cells changes channels Chapter chemical clinical complex compounds concentration decrease discussed disease dopamine dose drugs effects enzyme evidence example excretion factor function gene given glucose half-life heart hormone humans important increase inhibition inhibitors injection insulin interaction intracellular involved known less liver mainly mechanism mediators membrane metabolism molecules muscarinic myocardial infarction nerve terminals neurons norepinephrine normal occurs orally oxide pain particularly pathway patients peptide pharmacological phase plasma potential pressure prevent produce protein reactions receptors reduced regulation release renal response result role secretion selective shown similar smooth muscle specific stimulation structure studies substance sympathetic synthesis Table therapeutic therapy tissues transmission transmitter transport treat treatment tubocurarine unwanted effects uptake various vascular