PharmacologyChurchill Livingstone, 1995 - 855 pagine Pharmacology is now firmly established as a world wide best-selling and highly acclaimed textbook for medical and science students. Its successful approach emphasies the mechanisms by which drugs act and relates these to the overall pharmacological effects and clinical uses. It sets out for the reader a clear route from a molecular understanding of receptors and drug actions, to the therapeutic uses of the most important groups of drugs. For the Fifth Edition the text has been completely updated, incorporating significant new information published in the last few years, while obsolete material has been removed. New agents are fully discussed as well as extensions of basic knowledge which presage further drug development. Book jacket. |
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Pagina 271
... action potential takes a considerable time ( a few hundred milliseconds ) , and the refractory period outlasts the action potential . With fast - conducting fibers , recovery from inactivation of the sodium current is quick , and the ...
... action potential takes a considerable time ( a few hundred milliseconds ) , and the refractory period outlasts the action potential . With fast - conducting fibers , recovery from inactivation of the sodium current is quick , and the ...
Pagina 281
... Action potential Action potential с Vagal stimulation D 400 msec Fig . 13.8 Autonomic regulation of the heart beat . A and B. The effects of sympathetic stimulation and norepinephrine . C and D. The effects of parasympathetic ...
... Action potential Action potential с Vagal stimulation D 400 msec Fig . 13.8 Autonomic regulation of the heart beat . A and B. The effects of sympathetic stimulation and norepinephrine . C and D. The effects of parasympathetic ...
Pagina 289
... action potential in a train is only slightly reduced , but successive action potentials become progressively more blocked . With each action poten- tial , the opening of Na + channels allows further block to develop . If the preparation ...
... action potential in a train is only slightly reduced , but successive action potentials become progressively more blocked . With each action poten- tial , the opening of Na + channels allows further block to develop . If the preparation ...
Sommario
SECTION ONE 16 Hemostasis and Thrombosis | 331 |
The Respiratory System | 351 |
General Principles | 367 |
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acetylcholine acid action activity agents agonists anesthetics animals antagonists appears associated binding block blood body brain Ca2+ calcium cardiac cause cells central changes channels Chapter chemical clinical compounds concentration decrease dependence depression disease dopamine dose drugs effects enzyme evidence example excreted factor function given glucose half-life heart hormone important increase inhibition inhibitors injection insulin interaction involved known less liver mainly measured mechanism mediators membrane metabolism molecules myocardial infarction nerve neurons norepinephrine normal occurs orally organs pain pathway patients peptide Pharmacol pharmacological phase plasma potential pressure prevent produce protein reactions receptors reduced regulation release renal response result role secretion selective shown similar smooth muscle specific stimulation structure studies substances synthesis Table terminals therapeutic therapy tion tissues toxicity transmission transmitter treat treatment tubocurarine types unwanted effects usually various vascular